Dietary supplement for the treatment of gastritis and gastro-oesophageal reflux. Aligastril is a synergistic composition of active ingredients that combines the traditional use of natural antacids and alginates, with innovative phytotherapeutic active ingredients; it is also developed with the patented Lacursome® technology that optimizes the bioavailability of components on target tissues.
| Nutritional information | per maximum daily serving (2 sticks) |
| Sodium alginate | 2 g |
| Sodium bicarbonate | 2 g |
| Chamomile ES tit. 0.3% of which apigenin |
800 mg 2.4 mg |
| Chitosan | 500 mg |
| Melatonin | 1 mg |
| Nutritional information | per maximum daily serving (1 stick) |
| Sodium alginate | 500 mg |
| Chamomile ES tit. 0.3% of which apigenin |
400 mg 1.2 mg |
| Melatonin | 1 mg |
POWDER FORMAT:
Gluten-free oatmeal; Sodium alginate; Sodium hydrogen carbonate; Lemon juice powder; Acidity corrector: Citric acid; Chamomile (Matricaria chamomilla L.) flowers ES tit. 0.3% apigenin; Chitosan from mushrooms; Lemon flavor; Cocoa butter; Anti-caking agent: Silicon dioxide; Sweetener: Sucralose; Melatonin.
GEL FORMAT:
Water; Sodium alginate; Chamomile (Matricaria chamomilla L.) flowers ES tit. 0.3% apigenin; Acidity corrector: Sodium lactate sol. 60%, Citric acid; Lemon flavor; Preservative: Potassium sorbate; Sweetener: Sucralose; Melatonin.
Aligastril and Aligastril Gel: 20 sticks.

Heartburn, persistent acidity, a sense of heaviness that creeps in after meals. Those familiar with disorders such as gastritis or gastro-oesophageal reflux know how much these sensations can affect quality of life. Discomfort can begin with a simple gastric tightness, which gradually turns into a dull, diffuse pain, or explode into stinging twinges that rise toward the chest, sometimes mimicking pain of a cardiac nature.Stomach acidity can be a constant presence, an annoyance that accompanies every meal or appears unexpectedly in the middle of the night, disturbing rest and leaving a sour, bitter sensation in the mouth upon waking.
Gastro-oesophageal reflux, in particular, can manifest as a sensation of acid rising up along the esophagus, often accompanied by regurgitation, impaired breath, and an uncomfortable feeling of a knot in the throat. In some cases, gastro-oesophageal pain occurs after large or high-fat meals, with a retrosternal burning sensation that can last for several hours. It is not uncommon to feel pressure in the stomach that limits deep breathing, or mild but constant nausea that occurs without warning and hinders even the simplest daily activities.
Gastric pain often presents itself in an insidious way: a painful empty feeling, a persistent stinging in the upper abdomen, or a widespread irritation that worsens if one remains on an empty stomach or after morning coffee. In cases of gastritis, stomach pain may be intermittent or constant, becoming more acute with the change of seasons, with stress, or following disordered meals. Each person experiences these disorders with different nuances, but what they have in common is the interference with well-being, concentration, and the desire to sit at the table with serenity.
Aligastril meets these needs in two complementary formulations designed to suit different times of the day. The powder version, to be taken after meals, is suitable for regular, daily use. The gel stick, practical and ready to use, represents a pocket-sized ally, always at hand to manage the unexpected, even when out of the house. Both solutions are created to accompany those who experience these disorders, offering a simple gesture in response to a discomfort that should never be underestimated.
Gastric well-being is closely linked to a delicate balance between mucosal protective mechanisms and gastric acid secretion. When this balance is broken, acute gastritis occurs. More frequent causes of chronic gastritis, on the other hand, include infections from Helicobacter pylori, a bacterium capable of colonizing the gastric mucosa and profoundly altering its function, promoting chronic inflammatory processes and the breakdown of the stomach's natural protective defenses.
Gastro-oesophageal reflux is caused by the rise of the acidic contents of the stomach to the esophagus, which, not being equipped with the protective mucus, is irritated and attacked by the acidity, giving rise to the unpleasant sensations typical of reflux. In all these cases the combination of physiological, behavioral and environmental factors play a key role in the predisposition to the disorders and their evolution.
Seemingly innocuous daily behaviors can play a decisive role.Poor diet, characterized by irregular meals, excess fat, spicy foods, or carbonated beverages, adversely affects gastric emptying time and intra-abdominal pressure, making the onset of reflux and acute gastritis more frequent. Similarly, chronic stress is now considered a central element in the genesis of many gastrointestinal dysfunctions. Neuroendocrine mechanisms involved in stress responses can alter gastric motility, increase acid production, and impair the quality of protective mucus.
Equally,frequent or prolonged use of nonsteroidal anti-inflammatory drugs (NSAIDs) is a well-documented risk factor. These drugs, while widely used for the treatment of pain and inflammation, interfere with the production of prostaglandins, molecules critical for the regulation of protective mucus secretion. In the absence of adequate protection, the gastric mucosa is more exposed to the corrosive action of digestive acids.
Another non-negligible factor is bile reflux, a condition in which bile travels up from the stomach into the esophagus, contributing to irritation and changes in the gastro-oesophageal mucosa.Alcohol abuse can also compromise gastric health, both because of its direct effect on mucosal cells and because of its ability to increase acid secretion and alter the tone of the lower esophageal sphincter, facilitating the rise of gastric contents.
Finally, certain individual predispositions, such as acid hypersecretion or anatomical alterations of the stomach and esophagus, can facilitate the establishment of a fragile state of the mucosa, paving the way for inflammatory and degenerative processes. The complexity of these factors makes it clear how important it is to recognize and understand the dynamics that lead to the impairment of gastrointestinal balance.


The efficacy of Aligastril and Aligastril Gel is based on an integrated approach, capable of acting in synergy with the body's natural protective mechanisms to promote a functional rebalancing of the gastrointestinal system. Thanks to a unique combination of phytotherapeutic active ingredients and components with mechanical and regulatory action, the product exerts a comprehensive intervention designed to provide rapid and lasting relief while respecting digestive physiology.
Central to Aligastril's action is the formation of a protective barrier, which is achieved through the presence of sodium alginate. This substance, in contact with the acidic environment of the stomach, gives rise to a viscous gel that floats above the gastric contents, creating a mechanical anti-reflux effect. This physical layer helps to contain the rise of gastric juices and is an active protection of the gastric mucosa, reducing acid aggression and helping to restore tissue integrity.
The barrier effect ofalginate is complemented by the regulatory action of bicarbonate, which can locally neutralize excess acidity, thus helping to inhibit acid production and stabilize gastric pH. This buffering effect offers an immediate sense of rebalancing, especially at times when the gastric environment is particularly altered.
Aligastril's innovation is in the use of melatonin, traditionally used to regulate sleep-wake cycles but also crucial in gastric health. It has gastroprotective action in that it stimulates the production of bicarbonate by duodenal enterocytes, enhancing protection of the mucosa downstream of the stomach, and prostaglandins, which are critical in regulating the balance between mucus production and acidity. In addition, melatonin stimulates the contractile activity of the lower esophageal sphincter, whose function is impaired in gastro-oesophageal reflux. Finally, it stimulates the processes of angiogenesis and tissue repair, which are essential in cases of corrosion caused by stomach acid.
Finally, in Aligastril we find chamomile, rich in mucilage, polysaccharides and flavonoids, has anemollient and soothing action, while its antispasmodic component promotes a more regular and harmonious digestion, with an indirect effect on gas elimination and abdominal distension.
In Aligastril powder, we find the patented Lacursome® technology whereby the active ingredients of the product are encapsulated in lipid particles derived from cocoa butter, coated with pharmaceutical-grade, positively charged chitosan of fungal origin. Through this technology, the active ingredients are delivered to the gastric mucosa and adhere to it by electrostatic interaction with the negatively charged mucus layer.
Available in two distinct formats, Aligastril Powder and Aligastril Gel in ready-to-use sticks, the product adapts flexibly to different daily needs, offering valuable support against gastritis and reflux, in protecting the gastric mucosa and restoring a deep and natural feeling of well-being.
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